Search results for "CD11 Antigens"

showing 5 items of 5 documents

Deep venous thrombosis: Behaviour of the polymorphonuclear leukocyte integrin pattern at baseline and after in vitro activation

2005

In a group of 18 subjects with acute deep venous thrombosis (DVT), evidenced by clinical examination and echo-color-Doppler, we examined the phenotypical expression of the polymorphonuclear leukocyte (PMN) beta2-integrins (CD11a, CD11b, CD11c, CD18), obtained by using a flow cytofluorimeter. The evaluation was performed before and after in vitro activation (prolonged for 5 and 15 minutes) with 4-phorbol 12-myristate 13-acetate (PMA) and N-formyl-methionyl-leucyl-phenylalanine (fMLP). In DVT subjects, at baseline, the phenotypical expression of CD11b was decreased and that of CD11c was increased when compared with normal controls; no difference was found in CD11a and CD18 expression. In norm…

AdultMaleVenous ThrombosisIntegrinsSettore MED/09 - Medicina InternaCD11 AntigensNeutrophilsDeep venous thrombosis; polymorphonuclear leukocyte integrins; polymorphonuclear leukocyte activationMiddle AgedFlow CytometryNeutrophil ActivationDeep venous thrombosis polymorphonuclear leukocyte integrins polymorphonuclear leukocyte activationpolymorphonuclear leukocyte integrinsGene Expression RegulationCD18 AntigensCase-Control StudiesDeep venous thrombosisHumansTetradecanoylphorbol AcetateFemalepolymorphonuclear leukocyte activationAged
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Binding of monocytes from normolipidemic hyperglycemic patients with type 1 diabetes to endothelial cells is increased in vitro.

2009

Increased endothelial binding and emigration of monocytes play a dominant role in the pathogenesis of atherosclerosis in diabetes mellitus. Previous studies revealed that hyperlipidemia correlates with monocyte binding in vitro. The aim of this study was to characterize the monocyte-endothelial interaction of leucocytes of hyperglycemic patients with type 1 diabetes but lacking hyperlipidemia. We isolated monocytes from healthy controls and normolipidemic type 1 diabetes patients with elevated levels of HbA1c and quantified monocyte binding by an immunoilluminometric cell adhesion assay. Purity of isolated monocytes was at least 98%. Endothelial binding of monocytes from patients with type …

AdultMalemedicine.medical_specialtyArteriosclerosisEndocrinology Diabetes and MetabolismCell CountMonocytesPathogenesischemistry.chemical_compoundEndocrinologyInternal medicineDiabetes mellitusHyperlipidemiaInternal MedicinemedicineCell AdhesionHumansType 1 diabetesbusiness.industryCD11 AntigensImmunomagnetic SeparationMonocyteAntibodies MonoclonalGeneral Medicinemedicine.diseaseFlow CytometryIn vitroEndothelial stem cellEndocrinologymedicine.anatomical_structureDiabetes Mellitus Type 1GlucoseL-GlucosechemistryHyperglycemiaLuminescent MeasurementsMultivariate AnalysisRegression AnalysisFemaleEndothelium VascularbusinessExperimental and clinical endocrinologydiabetes : official journal, German Society of Endocrinology [and] German Diabetes Association
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Coincident airway exposure to low-potency allergen and cytomegalovirus sensitizes for allergic airway disease by viral activation of migratory dendri…

2019

Despite a broad cell-type tropism, cytomegalovirus (CMV) is an evidentially pulmonary pathogen. Predilection for the lungs is of medical relevance in immunocompromised recipients of hematopoietic cell transplantation, in whom interstitial CMV pneumonia is a frequent and, if left untreated, fatal clinical manifestation of human CMV infection. A conceivable contribution of CMV to airway diseases of other etiology is an issue that so far attracted little medical attention. As the route of primary CMV infection upon host-to-host transmission in early childhood involves airway mucosa, coincidence of CMV airway infection and exposure to airborne environmental antigens is almost unavoidable. For i…

Lung DiseasesPulmonologyMedizinCytomegalovirusImmunoglobulin EPathology and Laboratory MedicineWhite Blood CellsMiceAnimal CellsMedicine and Health SciencesCytotoxic T cellBiology (General)Enzyme-Linked ImmunoassaysImmune ResponseLung0303 health sciencesAntigen PresentationbiologyT Cells030302 biochemistry & molecular biologyAnimal Modelsrespiratory systemExperimental Organism SystemsFemalemedicine.symptomCellular TypesResearch ArticleQH301-705.5OvalbuminImmune CellsAntigen presentationImmunologyInflammationCytotoxic T cellsMouse ModelsResearch and Analysis MethodsMicrobiology03 medical and health sciencesSigns and SymptomsModel OrganismsTh2 CellsAntigenDiagnostic MedicineVirologyGeneticsmedicineHypersensitivityAnimalsT Helper CellsMolecular Biology TechniquesImmunoassaysMolecular Biology030304 developmental biologyInflammationBlood Cellsbusiness.industryCD11 AntigensBiology and Life SciencesCell BiologyDendritic CellsRC581-607Allergensrespiratory tract diseasesTransplantationMice Inbred C57BLOvalbuminDisease Models AnimalImmunologyRespiratory Infectionsbiology.proteinAnimal StudiesImmunologic TechniquesParasitologyVirus ActivationImmunologic diseases. AllergybusinessCD8CloningPLoS Pathogens
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Association of increased CCL5 and CXCL7 chemokine expression with neutrophil activation in severe stable COPD

2009

BACKGROUND: Increased numbers of activated neutrophils have been reported in the bronchial mucosa of patients with stable chronic obstructive pulmonary disease (COPD), particularly in severe disease. OBJECTIVES: To investigate the expression of neutrophilic chemokines and adhesion molecules in bronchial biopsies from patients with stable COPD of different severity (GOLD stages I-IV) compared with age-matched control subjects, smokers with normal lung function and never smokers. METHODS: The expression of CCL5, CXCL1, 5, 6, 7 and 8, CXCR1, CXCR2, CD11b and CD44 was measured in the bronchial mucosa using immunohistochemistry, confocal immunofluorescence, real-time quantitative polymerase chai…

MalePulmonary and Respiratory MedicineChemokinePathologymedicine.medical_specialtyCOPD neutrophils bronchial mucosa CCL5 CXCL7BronchiRespiratory MucosaGranulocyteNeutrophil ActivationCCL5Pulmonary Disease Chronic ObstructiveneutrophilsSubmucosaCOPDHumansMedicineCXC chemokine receptorsChemokine CCL5AgedCOPDbronchial mucosaCCL5biologySettore BIO/16 - Anatomia UmanaCD11 Antigensbusiness.industryCD44Epithelial CellsMiddle Agedrespiratory systemmedicine.diseaseRespiratory Function Testsrespiratory tract diseasesCXCL1Hyaluronan Receptorsmedicine.anatomical_structureAcute DiseaseImmunologyCXCL7biology.proteinFemaleLeukocyte ElastasebusinessCOPD; neutrophils; bronchial mucosa; CCL5; CXCL7Chemokines CXCCOPD CCL5CXCL7NEUTROPHILThorax
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Non-conventional forms of HLA-B27 are expressed in spondyloarthritis joints and gut tissue

2016

Objectives Human leukocyte antigen (HLA)-B27 (B27) is the strongest genetic factor associated with development of Ankylosing Spondylitis and other spondyloarthropathies (SpA), yet the role it plays in disease pathogenesis remains unclear. We investigated the expression of potentially pathogenic non-conventional heavy chain forms (NC) of B27 in synovial and intestinal tissues obtained from SpA patients. We also determined the presence of NC-B27 in joints, lymphoid and gastrointestinal tissue from B27 transgenic (TG1) rats with M.tuberculosis-induced SpA. Methods Expression of NC-B27 in human SpA joints and gut and in (21-3 × 283-2)F1 HLA-B27/Huβ2m rat tissue was determined by immunohistochem…

musculoskeletal diseasesalpha-DefensinsHLA-B27 transgenic rat modelGastrointestinal DiseasesCD8 AntigensImmunologyGene ExpressionArticleSpondyloarthropathieAnimalsHumansHLA class I free-heavy chainImmunology and AllergySpondylitis AnkylosingSpondyloarthropathiesskin and connective tissue diseasesHLA-B27 AntigenHLA-B27CD11 AntigensHistocompatibility Antigens Class ISynovial MembraneReceptors KIR3DL2Arthritis ExperimentalR1HLA class I free-heavy chainsRatsDisease Models AnimalSettore MED/16 - ReumatologiaHLA class I free-heavy chains; HLA-B27; HLA-B27 transgenic rat model; Spondyloarthropathies; Immunology and Allergy; ImmunologyBone RemodelingRats Transgenic
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